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PRELIMINARY REPORT

WILLIAM J. DIECKMANN, M.D.; HERBERT L. MICHEL, M.D.

Arch Intern Med. 1935;55(3):420-430.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

That vascular tension is an important factor in the mechanism of the toxemias of pregnancy was postulated many years ago. Cohnheim,¹ in 1880, seems to have been the first to express the view that albuninuria and anuria in eclampsia might be due to a spasm of the renal vessels. In 1918, Volhard² advanced the hypothesis that a generalized arterial spasm accounted for the hypertension, convulsions and renal symptoms in eclampsia. Heynemann³ also assumed that a general arterial spasm produced the symptoms. Various attempts to demonstrate vascular spasm have been more or less successful. Hinselmann⁴ (1924) noted the capillary changes in the finger-nail beds. He assumed that the capillary stasis and the dilatation were secondary to an arteriolar spasm. Subsequent observations of the nicking of retinal arteries and veins and the beading and the contractions of these vessels have indicated that the vascular spasm is probably general. . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Department of Obstetrics and Gynecology, the University of Chicago, and the Chicago Lying-In Hospital.

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