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PRESENCE OF PAIN FIBERS IN THE NERVE PLEXUS SURROUNDING THE CORONARY VESSELS

LOUIS N. KATZ, M.D.; WALTER MAYNE, B.S.; WILLIAM WEINSTEIN, B.S.

Arch Intern Med. 1935;55(5):760-772.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The most generally accepted explanation for the occurrence of pain in angina pectoris is the theory of cardiac ischemia. This theory is supported by several lines of evidence (the recent literature is reviewed by Keefer and Resnik¹ and Lewis²), one of which is based on animal experiments. This last type of evidence consists of the demonstration by Sutton and King,³ Pearcy, Priest and Van Allen⁴ and Sutton and Lueth⁵ of pain responses following occlusion of the coronary arteries in unanesthetized animals. These observers reported that pain was produced immediately on occlusion of the coronary arteries and that this pain ceased as soon as the vascular occlusion was released. Sutton⁶ stated: "In these experiments careful observations have been made on the effect of the temporary occlusion of the coronary arteries in unanaesthetized animals, with the conclusion that temporary occlusion results in immediate pain. It has been shown that this pain is . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Cardiovascular Laboratory, Department of Physiology, Michael Reese Hospital, and the Department of Physiology, University of Chicago.

Footnotes
Aided by the Frederick K. Babson Fund for the Study of Diseases of the Heart and Circulation, Michael Reese Hospital.

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