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I. RELATION TO URINARY ACIDITY

ELMER L. DeGOWIN, M.D.; HAROLD F. OSTERHAGEN, M.D.; MARIE ANDERSCH, Ph.D.

Arch Intern Med. 1937;59(3):432-444.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

From 1667, when the first blood transfusion was given to man by Jean Baptiste Denys,¹ until the discovery of hemagglutination by Landsteiner² in 1901 and the classification of blood groups by Jansk³ (1907) and Moss⁴ (1910), hemoglobinuria was accepted as a common accompaniment of the injection of blood intravenously. In many of the patients who showed hemoglobin in the urine renal insufficiency developed, and death followed. Since testing the donor's and the recipient's blood for agglutination has become common, the incidence of severe complications from blood transfusions has, fortunately, greatly diminished. Hesse and Filatov^18a reported a mortality of 1.3 per cent from 750 transfusions in Leningrad. They estimated that in general the mortality in various clinics was between 0.5 and 1.5 per cent. Tiber⁵ (1930) reported 2 deaths from 1,467 transfusions (0.013 per cent) at Bellevue Hospital. Witts⁶ described 3 deaths in 3,430 transfusions in Great . . . [Full Text PDF of this Article]

Author Affiliations
Associate in Internal Medicine, State University of Iowa IOWA CITY; Intern, Henry Ford Hospital DETROIT; Assistant Professor of Physiological Chemistry, Woman's Medical College PHILADELPHIA

From the Department of Internal Medicine, the State University of Iowa.

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