This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

DAVID A. RYTAND, M.D.

Arch Intern Med. 1937;59(5):837-847.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

That the pathogenesis of orthostatic albuminuria is poorly understood is attested by recent reviews.¹ The name orthostatic albuminuria is derived from a clinical symptom—the presence of an increased amount of protein in the urine while the subject is erect and its fall to a normal level (clinical absence) when he is supine. The object of this paper is to indicate the importance of determining the anatomic basis for this symptom by means of roentgenograms made after the injection of diodrast. The experience of my colleagues and myself in the first five cases in which this method was applied seems to indicate that orthostatic albuminuria arises from diverse anatomic anomalies and cannot be regarded as a distinct entity.

Two general theories may be considered seriously, the circulatory and the mechanical theory. The circulatory theory explains the anomaly on the following bases: First (a) in orthostatic albuminuria the amount of protein . . . [Full Text PDF of this Article]

Author Affiliations
SAN FRANCISCO

From the Department of Medicine, Stanford University School of Medicine.

Footnotes
Supported by a grant from the Rockefeller Fluid Research Fund.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?