This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

I. C. BRILL, M.D.; THOMAS D. ROBERTSON, M.D.

Arch Intern Med. 1937;60(6):1043-1057.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

At present two types of cor pulmonale are recognized—acute and chronic. The former is practically always the result of a single cause, pulmonary embolism; for a clearer understanding of its clinical manifestation one is indebted to the recent writings of Sylvester McGinn and Paul D. White.¹ In this form, except in those relatively rare instances in which the patient survives repeated attacks over a long period (two months or longer), there is rarely significant hypertrophy of the right ventricle, since the condition either is rapidly fatal or quickly results in complete recovery.

The chronic type of cor pulmonale is of manifold etiology. The most important causes of primary chronic cor pulmonale are mitral stenosis, extensive pulmonary fibrosis and marked emphysema secondary to asthma, chronic bronchitis or some other pulmonary disease. In 14 per cent of a series of one hundred patients with heart failure White² noted primary strain . . . [Full Text PDF of this Article]

Author Affiliations
PORTLAND, ORE.

From the Department of Medicine and the Department of Pathology of the University of Oregon Medical School and the Department of Pathology of St. Vincent's Hospital.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?