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A CORRELATION OF ELECTROCARDIOGRAPHIC AND PATHOLOGIC OBSERVATIONS

KURT BERLINER, M.D.; ARTHUR M. MASTER, M.D.

Arch Intern Med. 1938;61(1):39-59.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In recent years there has been a tendency to minimize the effects of valvular lesions and to regard infection of the myocardium as almost the sole determining factor in the course of rheumatic heart disease. This point of view¹ represents a reaction to the older, purely mechanical conception of valvular disease, and it may have been carried too far by some. The truth rests between the two extremes. As White² has pointed out:

With chronic healed valvular disease there need not be any trace of previous infection in the perfectly healthy muscle. Eventually the myocardium may become exhausted and fail . . . . In other words, it is the valve lesion, if well marked, and not the myocardial disease that eventually causes failure and death.

This profound effect of valvular lesions on the dynamics of the circulation is demonstrated in many ways. Fluoroscopic examination, for instance, shows significant changes in the . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Cardiographic Laboratory and the Medical Services of the Mount Sinai Hospital.

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