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VI. EXPIRATORY INFLATION DURING AIR HUNGER AND DYSPNEA PRODUCED BY PHYSICAL EXERTION IN NORMAL SUBJECTS AND IN PATIENTS WITH HEART DISEASE

JAMES A. GREENE, M.D.; L. W. SWANSON, M.D.

Arch Intern Med. 1938;61(5):720-725.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Extreme expiratory inflation was apparently of major importance in the production of air hunger and dyspnea in several of our patients with the effort syndrome. These observations suggested that enlargement of the expiratory volume of the chest might be a factor in the production of air hunger and dyspnea in patients with cardiac failure. In a previous study¹ the respirations were stimulated by reducing the oxygen or increasing the carbon dioxide content of the inspired air, both separately and simultaneously, and it was found that patients with cardiac failure were able to tolerate as great alterations of the inspired air as those tolerated by normal subjects. The former group, on the other hand, were unable to perform as much physical exercise as the normal subjects. These observations indicate that the respiratory stimulus produced by physical exertion is different from that produced by alteration of the inspired air. The purpose . . . [Full Text PDF of this Article]

Author Affiliations
IOWA CITY

From the Department of Internal Medicine, State University of Iowa College of Medicine.

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