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REPORT OF A CASE OF DISSECTING NECROTIC PNEUMONIA COMPLICATING PANCREATIC LITHIASIS

SYLVAN E. MOOLTEN, M.D.

Arch Intern Med. 1940;66(3):561-578.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The true nature of the lowered resistance to infection associated with diabetes mellitus is poorly understood despite a wealth of clinical and experimental information. Earlier writers¹ favored the view that it depended on hyperglycemia and saturation of the tissues with dextrose, since many micro-organisms, including diphtheroids, molds and cocci, had been shown to grow by preference in mediums containing sugar. More recent experiments, by Richardson,² Marble and his associates,³ Pillsbury and Kulchar⁴ and others, on the contrary, indicate that increased sugar content of blood and tissues is not significant in bringing about increased growth of bacteria in the body or in diminishing the resistance of the host. Although experimental staphylococcic cutaneous ulcers in dogs undergo rapid necrosis after large intraperitoneal injections of dextrose, this occurs only with solutions sufficiently hypertonic to have toxic effects, and these results can also be duplicated with hypertonic sodium chloride solution.⁴

The humoral mechanism . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Laboratory and Medical Service of St. Peter's Hospital, New Brunswick, N. J.

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