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DEFINITION, CLASSIFICATION AND PATHOGENESIS

PAOLO RAVENNA, M.D.

Arch Intern Med. 1940;66(4):879-892.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The Banti syndrome is still an unsolved problem. The question of cardinal importance is whether the splenomegaly is primary or secondary to portal or hepatic changes.

Banti¹ stated the belief that the trouble lay primarily in the spleen and that the portal and hepatic changes were dependent on the splenic ones. The idea that the splenomegaly was the consequence of circulatory disturbances was put forward by Dock and Warthin² and further developed with the description of the thrombophlebitic splenomegaly.³ This theory occupied a position of definite advantage when it was recognized that the structure of Banti splenomegaly was chiefly congestive⁴ and that the pressure in the splenic vein was highly increased.⁵ But these same signs of portal hypertension were found to be present when an obstructive factor was missing. This fact was responsible for the widest gap in the support of the theory of venous congestion as the causative agent . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

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