This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

ARCHIE H. BAGGENSTOSS, M.D.; EDWARD F. ROSENBERG, M.D.

Arch Intern Med. 1941;67(2):241-258.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Opinions as to the presence and nature of cardiac lesions associated with chronic infectious (rheumatoid) arthritis have been varied. Fuller¹ and Garrod² have stated that, as a rule, the heart is not injured in rheumatoid arthritis. Fuller stated that he had never seen a case in which he could attribute pericarditis or endocarditis to rheumatoid arthritis. Garrod observed that if patients with rheumatoid arthritis have valvular insufficiency, one can assume a previous attack of rheumatic fever has been the cause of the lesion.

Strümpell³ and Pribram⁴ stated that in rheumatoid arthritis of slow, insidious onset cardiac involvement does not occur, but when the disease is ushered in by an acute febrile attack of arthritis the heart is often implicated.

Barjon,⁵ on the other hand, estimated that the number of cardiac complications is just as high in the cases of rheumatoid arthritis with chronic onset as . . . [Full Text PDF of this Article]

Author Affiliations
ROCHESTER, MINN.

From the Section on Pathology, the Mayo Clinic (Dr. Baggenstoss).

Footnotes
Fellow in Medicine, the Mayo Foundation.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?