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MICHAEL SOMOGYI, Ph.D.

Arch Intern Med. 1941;67(3):665-679.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The presence of diastase¹ in human blood was first noted in 1846² and subsequently the enzyme was found to be a normal constituent of mammalian blood in general. In 1863 the enzyme was shown to be present also in urine, and its origin was traced to the blood.³ Eventually diastase was detected in lymph,⁴ in pericardial fluid^4b and in feces.⁵ Hepatic bile is almost completely devoid of diastase,⁶ but the enzyme can be detected in the bile when the diastase content of blood is greatly increased by ligation of the pancreatic ducts.⁷ Bile from the common bile duct and the gallbladder may contain appreciable amounts of diastase as the result of pancreatic reflux.⁸ Assertions that measurable amounts of diastase are present in the cerebrospinal fluid,⁹ the liver and the kidneys¹⁰ are open to doubt. In this laboratory it was not possible to detect diastase in the . . . [Full Text PDF of this Article]

Author Affiliations
ST. LOUIS

From the laboratory of the Jewish Hospital.