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THERAPY OF B. FRIEDLÄNDER B PNEUMONIA

ELY PERLMAN, M.D.; JESSE G. M. BULLOWA, M.D.

Arch Intern Med. 1941;67(5):907-920.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In 1882 Friedländer¹ described a gram-negative, encapsulated bacillus which was at first regarded as the cause of pneumonia. Four years later Weichselbaum² demonstrated that Diplococcus pneumoniae was the more frequent etiologic agent. Recent extensive studies show that Bacillus Friedländer causes about 0.5 to 4.0 per cent of all the pneumonias and accordingly will be encountered several times a year in any large medical service.

In 1925 Avery, Heidelberger and Goebel³ demonstrated the chemical and immunologic relationship of certain strains of this bacillus to Pneumococcus type II. The following year Julianelle⁴ classified a number of strains of B. Friedländer by immunologic methods under types A, B and C with a heterologous group X. There are now available the Friedländer bacillus A and the Friedländer bacillus B serum for identification of strains by the Neufeld technic, but the B strain also swells with the type II pneumococcus typing . . . [Full Text PDF of this Article]

Author Affiliations
Clinical Professor of Medicine, New York University College of Medicine; Visiting Physician, Harlem Hospital NEW YORK

From the Medical Service, Harlem Hospital, Department of Hospitals, and the Littauer Pneumonia Research Fund of New York University College of Medicine.

Footnotes
Littauer Fellow in Pneumonia Research.

This study received additional financial support from the Metropolitan Life Insurance Company and from Mr. Bernard M. Baruch. Mr. Bernard M. Baruch Jr., Miss Belle N. Baruch and Mrs. H. Robert Samstag.

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