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LIEUTENANT EDWARD H. REISNER, Jr.

Arch Intern Med. 1943;71(2):230-255.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Acute hemolytic anemias due to autoantibody reactions have been observed in human beings¹ and induced experimentally in animals.² Hematologic examination of the experimental animals has revealed a fairly consistent pattern of morphologic change. During the initial phase of hemolysis, the mean cell diameter decreases and the mean cell volume increases, the cells assuming a spheroid shape. After several days the mean diameter rises above normal with the simultaneous appearance of macrocytes, nucleated red cells and increased numbers of reticulocytes. At this time the Price-Jones curve is frequently bimodal, and Dameshek has stressed this point as being of diagnostic import in such anemias. The recent work of Levine and his associates³ has shown that about 90 per cent of infants with erythroblastosis foetalis have an antigen, designated as Rh, in their blood cells which is inherited from the father, probably as a mendelian dominant character, and which the . . . [Full Text PDF of this Article]

Author Affiliations
MEDICAL CORPS, ARMY OF THE UNITED STATES

From the New York Hospital and the Department of Pediatrics, Cornell University Medical College.

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