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LIEUTENANT R. S. MEGIBOW; L. N. KATZ, M.D.; PRIVATE M. FEINSTEIN

Arch Intern Med. 1943;71(4):536-546.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Previous investigators clearly demonstrated that the invariable response to diffuse embolism of pulmonary arterioles and capillaries is primarily that of tachypnea,¹ while the response following diffuse embolism of the moderately sized pulmonary arteries is characterized by both tachypnea and dyspnea.² It was concluded³ that the underlying mechanism of these respiratory changes was different in that the rapid respiratory rate subsequent to the larger emboli was due apparently to anoxemia while that following the smaller emboli was the result of pulmonary congestion and edema. The latter appeared to operate by reflex action on the respiratory center by stimulation of the vagal nerve endings in the lungs.⁴

During the investigation of the cardiovascular dynamics of and the mechanism of death from experimental pulmonary embolism⁵ we had occasion to confirm the essential differences in the character of respiration which follows moderately sized and miliary embolism. During the course . . . [Full Text PDF of this Article]

Author Affiliations
MEDICAL CORPS, ARMY OF THE UNITED STATES; CHICAGO; SIGNAL CORPS, ARMY OF THE UNITED STATES

From the Cardiovascular Department. Michael Reese Hospital.

Footnotes
Aided by the A. D. Nast and the Emil and Fanny Wedeles Fund for Cardiovascular Research.

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