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T. ADDIS, M.D.; EVALYN BARRETT; W. LEW; L. J. POO; D. W. YUEN

Arch Intern Med. 1946;77(3):254-259.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

IN cases of shock there may be a sudden loss of effective renal tissue, because for a time the kidney does not get enough blood; more sustained deprivation sometimes occurs after transfusion reactions; there are instances of anatomic as well as functional loss, as when part of the kidney is destroyed by shell fragments, and in glomerular nephritis and symmetric necrosis of the cortex the majority of the nephrons in a short space of time may become functionless and many may be irrevocably destroyed. For each of these situations there is a specific treatment dependent on knowledge of the causative mechanism, but in all, except the most transitory instances of functional loss, we believe that the work demanded from the remaining renal tissue should be reduced. The work of the kidney consists almost wholly in the reabsorption of water from a concentrated solution of urea in the tubules into a . . . [Full Text PDF of this Article]

Author Affiliations
SAN FRANCISCO

From the Department of Medicine, Stanford University School of Medicine.

Footnotes
The work described in this paper was done under a contract, recommended by the Committee on Medical Research, between the Office of Scientific Research and Development and Stanford University.

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