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J. A. ROSENKRANTZ, M.D.; CLARE MARSHALL, B.A.

Arch Intern Med. 1947;80(1):81-88.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

IN RECENT years great strides have been made in the direction of determination of the pathogenesis of hypertensive vascular disease. However, there is conflicting opinion concerning the relation between basal heat production and hypertensive vascular disease.¹ Clinically, an elevated basal metabolic rate has been noted in patients with hypertension. Mountain and his co-workers^1h reported that there was a direct correlation between basal heat production and the height of the blood pressure; others² failed to demonstrate this relation. The exact mechanism underlying these metabolic changes has not been definitely proved. Some investigators³ have indicated that an interrelationship among the brain, the thyroid and the adrenal sympathetic system exists. Hayasaka^1f concluded that the basal metabolic rate remains unchanged in hypertensive patients with normal renal function but becomes elevated in those with renal insufficiency.

Because of these opposing viewpoints, it seemed of real value to reinvestigate the entire problem. In . . . [Full Text PDF of this Article]

Author Affiliations
Research Assistant in Medicine, New York Post-Graduate Medical School and Hospital; NEW YORK

From the Department of Medicine and the Respiration Laboratory, New York Post-Graduate Medical School and Hospital.

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