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G. LILJESTRAND, M.D.

Arch Intern Med. 1948;81(2):162-172.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

SINCE Hering's discovery,¹ about twenty years ago, that increased pressure in the carotid sinus reflexly produces bradycardia and arteral hypotension, a great deal of work has been done in the regulation of systemic arterial blood pressure. It has been proved, especially by the work of Heymans and his associates,^1a that the sinus and aortic regions are of paramount importance in this connection; on the one hand, increased arterial pressure stimulates pressosensitive endings of the sinus and aortic nerves and provokes inhibition of the cardiac and vasomotor centers, and, on the other hand, the lowering of the oxygen and the rise of the carbon dioxide tension in the arterial blood stimulate chemoreceptors and from them, by way of a reflex, also the centers mentioned. Having been for some time engaged in studies on these questions it appeared to my colleague, Professor von Euler, and me² that it . . . [Full Text PDF of this Article]

Author Affiliations
STOCKHOLM, SWEDEN

Footnotes
Read before the Section on Pathology and Physiology at the Ninety-Sixth Annual Session of the American Medical Association, Atlantic City, N. J., June 11, 1947.

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