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MOGENS BJØRNEBOE, M.D.; CLAUS BRUN, M.D.; FLEMMING RAASCHOU, M.D.

Arch Intern Med. 1949;83(5):539-546.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

DURING the course of the epidemic of acute infectious hepatitis occurring in Denmark at the time of this report, several cases of chronic hepatitis were noted in elderly women.¹ The characteristics of this form of hepatitis seem to be a grave, usually fatal, course, during which considerable changes take place in the plasma proteins: The value for serum albumin falls and that for serum globulin rises, and symptoms develop suggesting a low colloid osmotic pressure, viz., ascites and edema. The works which so far have dealt with the relation of colloid osmotic pressure to chronic hepatitis are few; moreover, the results published do not agree.² Myers and Keefer,³ Tumen and Bockus⁴ and Post and Patek⁵ noted a decrease in plasma albumin and accordingly suggested low colloid osmotic pressure as a cause of edema; however, the pressure was not measured directly. Consequently, we thought it justifiable to take advantage of the . . . [Full Text PDF of this Article]

Author Affiliations
COPENHAGEN, DENMARK

From the Third Department, Kommune Hospital; Dr. Poul Iversen, Chief.

Footnotes
This work was carried out with support from the King Christian X Fund.

Read before the Twentieth Scandinavian Congress for Internal Medicine, Göteborg, Sweden, June, 1946.

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