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Evidence for a Common Etiology

ROBERT S. EVANS, M.D.; KAZUYUKI TAKAHASHI, M.D.; ROSE T. DUANE, A.B.; ROSE PAYNE, Ph.D.; CHI-KONG LIU, M.D.

AMA Arch Intern Med. 1951;87(1):48-65.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

MANY EXPLANATIONS have been advanced for the low thrombocyte (platelet) count in primary or idiopathic thrombocytopenic purpura, but so far all theories lack substantial proof. The disease is somewhat commoner in females and occurs more frequently in the first half of life, although it has been seen in persons of all age groups. A familial history of easy bruising and prolonged bleeding is frequently mentioned in case histories, but few hematological data are offered to support the supposition that thrombocytopenia has occurred in other members of the family. Reports of transitory thrombocytopenia in infants of women with thrombocytopenic purpura are of particular interest, since these cases suggest that an agent producing the thrombocytopenia is transmissible through the placenta.¹ Both increased destruction and decreased formation of thrombocytes have been advanced as causes of primary thrombocytopenia. Since there is no technic comparable to the reticulocyte count for estimating the rate of . . . [Full Text PDF of this Article]

Author Affiliations
SAN FRANCISCO

From the Department of Medicine, Stanford University School of Medicine.

Footnotes
This work was supported by a grant from the United States Public Health Service.

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