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MYOCARDIAL INFARCTION WITH AND WITHOUT ACUTE CORONARY OCCLUSIONA Pathologic Study
R. DREW MILLER, M.D.;
HOWARD B. BURCHELL, M.D.;
JESSE E. EDWARDS, M.D.
AMA Arch Intern Med. 1951;88(5):597-604.
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THE CONCEPT of inadequate coronary blood flow resulting from chronic atherosclerotic narrowing of the coronary arteries as a cause of myocardial infarction without acute occlusion has become rather generally recognized.1 Among the first pathologic observations of acute myocardial infarction without the presence of an acute coronary occlusion were the studies of Büchner.2 Blumgart, Schlesinger, and Zoll3 have stressed the fact that coronary thrombosis or occlusion does not always produce myocardial infarction and, conversely, that acute myocardial infarction may occur in the absence of acute coronary occlusion. They have demonstrated this particularly by the technic of studying the coronary arteries after injection of radiopaque compounds. Experimentally, both the severity and the duration of the hypoxia of the myocardium occurring with coronary insufficiency would seem to determine whether the changes will be transient and reversible or be characterized by necrosis of the myocardium.4
The primary purpose of this
. . . [Full Text PDF of this Article]
Author Affiliations
ROCHESTER, MINN.
Dr. Miller is Fellow in Medicine, Mayo Foundation. Dr. Burchell is from the Division of Medicine, and Dr. Edwards, from the Section on Pathologic Anatomy, Mayo Clinic.
Footnotes
This paper is an abridgment of a thesis submitted by Dr. Miller to the Faculty of the Graduate School of the University of Minnesota in partial fulfilment of the requirements for the degree of Master of Science in Medicine.
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