This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

ALEXANDER ALTSCHUL, M.D.; PEARL D. FOSTER, M.D.; SAMUEL S. PALEY, M.D.; LEONA TURNER, M.D.

AMA Arch Intern Med. 1952;89(1):24-31.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

HEPATITIS has become an important problem in the practice of medicine during the past two decades. Its incidence is increasing. Originally, attention was focused upon the liver because of damage produced by hepatotoxic agents in therapy, as well as in industry. Of the drugs and chemicals responsible for liver damage, the following may be cited: cinchopen, sulfonamides, bismuth, arsenicals, colloidal gold, quinine, and quinacrine hydrochloride (atabrine^® dihydrochloride).¹ Among the industrial poisons are carbon tetrachloride, which is outstanding, trinitrotoluene, tetrachloroethylene, and phosphorus. Chloroform and ether are other chemical poisons which may act as toxic irritants on the liver.²

Many infections are capable of producing acute hepatic injury, i. e., virus pneumonia, infectious mononucleosis,³ acute brucellosis, malaria,⁴ and amebiasis.

During World War II a great number of cases of hepatitis appeared in epidemic form, and it was discovered that the etiological factor in many of them wasa virus with a definite incubation . . . [Full Text PDF of this Article]

Author Affiliations
NEW YORK

From the Medical Service of Harlem Hospital.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?