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STUDIES OF FLUID, ELECTROLYTE, AND NITROGEN BALANCE IN ACUTE RENAL INSUFFICIENCY
LLOYD T. ISERI, M.D.;
THOMAS M. BATCHELOR, M.D.;
ALBERT J. BOYLE, M.D.;
GORDON B. MYERS, M.D.
AMA Arch Intern Med. 1952;89(2):188-215.
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ACUTE renal insufficiency, including the syndrome of lower nephron nephrosis, is characterized not only by retention of nitrogenous end-products but also by profound disturbance in fluid and electrolyte metabolism. Thorough understanding of the underlying pathophysiology is essential for the correct management of these patients. Bywaters1 renewed interest in the syndrome of acute renal insufficiency with his description of azotemia following crushing injuries. The pathologic changes, the pathogenesis, and the clinical picture have been summarized recently by Burch and Ray,2 Muirhead,3 Strauss,4 and Lucké.5
Since the maintenance of fluid and electrolyte equilibrium during the critical oliguric and early diuretic phase of lower nephron syndrome appears distinctly to influence the fatality rate, an effort has been made to study the nature of disturbances in these equilibria. Complete metabolic studies of sodium, potassium, chloride, nitrogen, and water balance were made in 5 cases of severe acute renal insufficiency for periods of 6 to 23
. . . [Full Text PDF of this Article]
Author Affiliations
DETROIT
From the Departments of Medicine and Chemistry, Wayne University, and the Department of Medicine, City of Detroit Receiving Hospital.
Footnotes
This study was supported by grants from the National Heart Institute and the Michigan Heart Association.
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