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ROLAND V. MURPHY, M.D.; EUGENE W. COFFMAN, M.D.; BENJAMIN H. PRINGLE, Ph.D.; LLOYD T. ISERI, M.D.

AMA Arch Intern Med. 1952;90(6):750-762.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

SINCE THE term "salt-losing nephritis" was introduced by Thorn, Koepf, and Clinton¹ in 1944, in their study of two cases simulating adrenal cortex insufficiency, four additional cases have been reported.² An associated tendency toward excessive loss of potassium was found in one case,^2c and electrocardiographic evidence of hyperpotassemia was seen in another.^2d The present report is concerned with a case of salt-losing nephritis, followed for two years, in which sodium was lost and potassium was retained in excess quantities.

REPORT OF A CASE

J. B., a 30-year-old man, was first admitted to the hospital on June 19, 1950, with complaints of weakness for one year; periodic vomiting before breakfast and intermittent bleeding from the gums for three months, and occasional hematemesis, nocturia, paresthesia of hands and feet, and cramping of the calf muscles for six weeks. The patient was anorexic and had been vomiting frequently just prior to admission.

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Author Affiliations
DETROIT; DEARBORN, MICH.; DETROIT

From the Departments of Medicine, Veterans Administration Hospital, Dearborn, Mich., and Wayne University College of Medicine, Detroit.

Footnotes
This study was supported in part by grants from the National Heart Institute and the Michigan Heart Association.

Reviewed in the Veterans Administration and published with the approval of the Chief Medical Director. The statements and conclusions published by the authors are the result of their own study and do not necessarily reflect the opinion or policy of the Veterans Administration.

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