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FIRST LIEUTENANT WILLIAM H. WAUGH

AMA Arch Intern Med. 1954;93(2):299-303.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

IODIDE reactions are pernicious. Rich¹ described a case in which iodide appeared to cause the destructive lesions of necrotizing arteritis. Deaths due to iodide have been observed, either from angioneurotic laryngeal edema or from exfoliative dermatitis.* In the past therapy for iodide reactions has consisted largely of symptomatic measures, forced fluid, and chloride administration, preferably of the ammonium rather than the sodium salt.

Recently, two reports have appeared on the effect of corticotropin on iodide hypersensitivity. Carey and co-workers⁴ described a case of severe iodism, with angioneurotic edema, submaxillary adenitis, and extensive exfoliative dermatitis, in which marked improvement occurred within 48 hours after institution of corticotropin therapy. Cessation of therapy resulted in a recurrence of the exfoliation, which again appeared to be controlled by corticotropin administration. Theodos⁵ reported the case of a patient known to be allergic to iodide, in which the ingestion of about 10 ml. . . . [Full Text PDF of this Article]

Author Affiliations
UNITED STATES AIR FORCE

From the Department of Medicine, United States Air Force Hospital, Nagoya, Honshu, Japan.

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