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JOHN R. HOGNESS, M.D.; JAMES M. BURNELL, M.D.

AMA Arch Intern Med. 1954;93(3):355-366.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

PROGRESSIVE renal failure, when present in such varying diseases as chronic glomerulonephritis, chronic pyelonephritis, hydronephrosis, arteriolonephrosclerosis, and polycystic kidneys, is usually characterized by common clinical manifestations. Hypertension, retinopathy, edema, anemia, nitrogen retention, albuminuria, and cylindruria are usually present at some time during the course of the disease. Exceptions occur, of course, but in general the late manifestations are quite constant, regardless of the underlying renal process.

This paper deals with four cases which deviated consistently from this typical clinical pattern. In all these cases the kidneys were found to be small and contracted and to contain numerous medullary cysts. The clinical picture was characterized by insidious uremia and anemia alone, unaccompanied by the other stigmata of progressive renal failure. Three of these were taken from the files of the Presbyterian Hospital and one from the files of Dr. Warfield T. Longcope. In addition, reference is made to two cases from . . . [Full Text PDF of this Article]

Author Affiliations
SEATTLE

From the Departments of Medicine and Pathology, College of Physicians and Surgeons, Columbia University, and the Presbyterian Hospital, New York.

Footnotes
Present address of Dr. Hogness and Dr. Burnell: Department of Medicine, University of Washington.

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