This Article  • References  • Full text PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citing articles on HighWire  • Contact me when this article is cited  Related Content  • Similar articles in this journal  Social Bookmarking   What's this?

Transient Hypertension and Tachycardia Caused by Disruption of the Carotid Sinus; Review of Buffer Nerve Mechanism

PAUL KEZDI, M.D.

AMA Arch Intern Med. 1954;94(1):122-130.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Normal blood pressure has a very sensitive adaptability to the demands of the circulation mediated through the reflex system of the carotid sinus and aortic nerves. Physiologists envision normal blood pressure as a balance between two forces: intrinsic vasomotor tone on one hand and constant counteraction of the sinoaortic regulatory system on the other. Superimposed upon the vasomotor center in the medulla there are higher coordination centers of the circulation in the hypothalamus. W. R. Hess distinguishes two parts of the hypothalamic circulatory center, a posterior and an anterior part. In the cat, electrical stimulation of the former leads to blood pressure rise and tachycardia; stimulation of the latter leads to blood pressure fall and pulse rate decrease.¹ Bronk and co-workers found that, depending on the frequency of stimulation, the same area can give either result.² They concluded that hypothalamic activity regulates the effectiveness of the afferent inhibitor . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Department of Medicine, Wesley Memorial Hospital and Northwestern University Medical School.

CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?