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ALBERT R. ALLEN, M.D.

AMA Arch Intern Med. 1956;98(4):463-466.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

With the advent of long-term chemotherapy ⁶ and the resultant conversion of a large percentage of patients' sputums and gastric cultures, there were many optimistic opinions that the present antituberculous drugs would cure tuberculosis. D'Esopo and co-workers ⁷ reported that when these patients whose cultures had converted on streptomycin and PAS were resected, it was difficult to grow tubercle bacilli from the resected specimens. However, acid-fast rods were present in a large number of these specimens, so the question arose: Are these tubercle bacilli dead or merely dormant?¹¹ By six months after chemotherapy was stopped a significant percentage of the negative nonresected patients with open cavities had reactivated.¹² On June 1, 1952, we began our present treatment regimen, consisting of streptomycin, aminosalicylic acid, and isoniazid used concurrently and continuously from admission to discharge, with early resectional surgery when indicated.¹ When digested portions of our first 22 resected . . . [Full Text PDF of this Article]

Author Affiliations
Selah, Wash.

Medical Director and Superintendent, Central Washington Tuberculosis Hospital.

Footnotes
Submitted for publication April 14, 1956.

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