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An Explanation of the Transient Stroke

ELIOT CORDAY, M.D.; SANFORD ROTHENBERG, M.D.; STANLEY MARTIN WEINER, M.D.

AMA Arch Intern Med. 1956;98(6):683-690.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

Acute cerebral vascular insufficiency may be defined physiologically as a deficiency of cerebral arterial blood flow resulting from an inadequate systemic arterial blood pressure or impairment of the cardiac output.¹ This condition is often transient and correctible, although the resulting deleterious effect upon the brain may or may not be permanent. The deficit in cerebral blood flow may involve the whole brain, or it may be localized. When it is generalized, the well-known symptoms of syncope, generalized grand mal seizures, etc. may occur. It has not been clearly recognized, however, that focal cerebral manifestations, such as hemiplegia, hemisensory disturbances, and Jacksonian seizures, are not uncommonly the result of localized cerebral vascular insufficiency. In the past, such disorders when permanent have been attributed to cerebral hemorrhage, thrombosis, or embolism. When focal cerebral signs have been transient, they have usually been considered a result of localized cerebral vascular spasm. It is . . . [Full Text PDF of this Article]

Author Affiliations
Los Angeles; Denver

Footnotes
Submitted for publication June 20, 1956.

Institute for Medical Research, Cedars of Lebanon Hospital.

This paper was discussed by Dr. Geza De Takats, Chicago.

This study was aided by a research grant from the Beneficial Standard Life Insurance Company, Los Angeles.

Read before the Section on Internal Medicine at the 105th Annual Meeting of the American Medical Association, Chicago, June 12, 1956.

Department of Medicine, University of California Medical Center, Los Angeles (Dr. Corday); former resident in Medicine, Cedars of Lebanon Hospital (Dr. Weiner).

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